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Nature Neuroscience. Zinc binds at Recent studies have article source that source, along with the non-stimulants atomoxetine and extended-release guanfacine, are continuously effective for more than 2-year treatment periods with few and tolerable adverse effects. Journal of Attention Disorders. The prevalence of this withdrawal click is extremely common Cantwell ; Gossop with Views Read View source View history. Archived from the original on 2 October Click the following article is used to treat attention deficit hyperactivity disorder ADHD. NBC Amphetaminöl.

Pharmacodynamics of amphetamine in a dopamine neuron v t e. Metabolic pathways of amphetamine in humans [sources 16]. Benzoic acid.

Hippuric acid. Para- Hydroxylation. Beta- Hydroxylation. DBH [note 17]. Oxidative Deamination. Glycine Conjugation. Further information on related compounds: Trace amine.

Racemic amphetamine. Amphetamine hydrochloride left bowl Phenylnitropropene right cups. For a more comprehensive list, see Substituted amphetamine.

Main article: History and culture of substituted amphetamines. See Mixed amphetamine salts for more information about the mixture, and this section for information about the various mixtures of amphetamine enantiomers currently marketed.

USFDA contraindications are not necessarily intended to limit medical practice but limit claims by pharmaceutical companies.

Due to pharmacological differences between these medications e. Text color Transcription factors. Retrieved 22 December Prescriber's Guide: Stahl's Essential Psychopharmacology 6th ed.

Retrieved 5 August Journal of Psychopharmacology. The intravenous use of d-amphetamine and other stimulants still pose major safety risks to the individuals indulging in this practice.

Some of this intravenous abuse is derived from the diversion of ampoules of d-amphetamine, which are still occasionally prescribed in the UK for the control of severe narcolepsy and other disorders of excessive sedation.

For these reasons, observations of dependence and abuse of prescription d-amphetamine are rare in clinical practice, and this stimulant can even be prescribed to people with a history of drug abuse provided certain controls, such as daily pick-ups of prescriptions, are put in place Jasinski and Krishnan, b.

University of Alberta. Retrieved 5 November United States Food and Drug Administration. Shire US Inc. December Retrieved 30 December Journal of Pharmaceutical and Biomedical Analysis.

Retrieved 21 January In Millichap JG ed. Table 9. Postgraduate Medicine. Hazardous Substances Data Bank. Archived from the original on 2 October Retrieved 2 October Duration of effect varies depending on agent and urine pH.

Excretion is enhanced in more acidic urine. Amphetamines are distributed into most body tissues with high concentrations occurring in the brain and CSF.

Foye's principles of medicinal chemistry 7th ed. The simplest unsubstituted phenylisopropylamine, 1-phenylaminopropane, or amphetamine, serves as a common structural template for hallucinogens and psychostimulants.

Amphetamine produces central stimulant, anorectic, and sympathomimetic actions, and it is the prototype member of this class The phase 1 metabolism of amphetamine analogs is catalyzed by two systems: cytochrome P and flavin monooxygenase.

Amphetamine can also undergo aromatic hydroxylation to p -hydroxyamphetamine. Stereochemical course of the reaction" PDF. Journal of Biological Chemistry.

Retrieved 6 November Journal of Pharmacology and Experimental Therapeutics. In Klee H ed. Amsterdam, Netherlands: Harwood Academic Publishers.

Retrieved 1 December Amphetamine, in the singular form, properly applies to the racemate of 2-aminophenylpropane.

In its broadest context, however, the term [ amphetamines ] can even embrace a large number of structurally and pharmacologically related substances.

PubChem Compound Database. Retrieved 9 November Royal Society of Chemistry. Retrieved 13 October Retrieved 17 April Emergency Medicine Australasia.

International Union of Pure and Applied Chemistry. Archived from the original on 17 March Retrieved 14 March One of a pair of molecular entities which are mirror images of each other and non-superposable.

Therapeutic relatively low doses of psychostimulants, such as methylphenidate and amphetamine, improve performance on working memory tasks both in normal subjects and those with ADHD.

Thus, stimulants improve performance on effortful but tedious tasks Beyond these general permissive effects, dopamine acting via D1 receptors and norepinephrine acting at several receptors can, at optimal levels, enhance working memory and aspects of attention.

Primary Care: Clinics in Office Practice. Amphetamines and caffeine are stimulants that increase alertness, improve focus, decrease reaction time, and delay fatigue, allowing for an increased intensity and duration of training Journal of the History of Medicine and Allied Sciences.

However the firm happened to discover the drug, SKF first packaged it as an inhaler so as to exploit the base's volatility and, after sponsoring some trials by East Coast otolaryngological specialists, began to advertise the Benzedrine Inhaler as a decongestant in late United Nations Treaty Collection.

United Nations. Archived from the original on 31 March Retrieved 11 November Stimulant misuse appears to occur both for performance enhancement and their euphorogenic effects, the latter being related to the intrinsic properties of the stimulants e.

Although useful in the treatment of ADHD, stimulants are controlled II substances with a history of preclinical and human studies showing potential abuse liability.

Medical Subject Headings. United States National Library of Medicine. Retrieved 16 December World Health Organization.

In principle, INNs are selected only for the active part of the molecule which is usually the base, acid or alcohol. In some cases, however, the active molecules need to be expanded for various reasons, such as formulation purposes, bioavailability or absorption rate.

In the experts designated for the selection of INN decided to adopt a new policy for naming such molecules. In future, names for different salts or esters of the same active substance should differ only with regard to the inactive moiety of the molecule.

Arbor Pharmaceuticals, LLC. National Drug Code Directory. Archived from the original on 16 December Journal of Neurochemistry.

Drug and Alcohol Dependence. When considered together with the rapidly growing literature in the field a compelling case emerges in support of developing TAAR1-selective agonists as medications for preventing relapse to psychostimulant abuse.

Shoptaw SJ, Ali R ed. A minority of individuals who use amphetamines develop full-blown psychosis requiring care at emergency departments or psychiatric hospitals.

In such cases, symptoms of amphetamine psychosis commonly include paranoid and persecutory delusions as well as auditory and visual hallucinations in the presence of extreme agitation.

Findings from one trial indicate use of antipsychotic medications effectively resolves symptoms of acute amphetamine psychosis. BMC Psychiatry.

In these studies, amphetamine was given in consecutively higher doses until psychosis was precipitated, often after — mg of amphetamine Archived from the original PDF on 3 November Retrieved 2 November CNS Drugs.

Several other studies, [] including a meta-analytic review [98] and a retrospective study, [97] suggested that stimulant therapy in childhood is associated with a reduced risk of subsequent substance use, cigarette smoking and alcohol use disorders.

Recent studies have demonstrated that stimulants, along with the non-stimulants atomoxetine and extended-release guanfacine, are continuously effective for more than 2-year treatment periods with few and tolerable adverse effects.

The effectiveness of long-term therapy includes not only the core symptoms of ADHD, but also improved quality of life and academic achievements.

The most concerning short-term adverse effects of stimulants, such as elevated blood pressure and heart rate, waned in long-term follow-up studies.

The current data do not support the potential impact of stimulants on the worsening or development of tics or substance abuse into adulthood.

Such agents also have important therapeutic uses; cocaine, for example, is used as a local anesthetic Chapter 2 , and amphetamines and methylphenidate are used in low doses to treat attention deficit hyperactivity disorder and in higher doses to treat narcolepsy Chapter Despite their clinical uses, these drugs are strongly reinforcing, and their long-term use at high doses is linked with potential addiction, especially when they are rapidly administered or when high-potency forms are given.

Current Medical Research and Opinion. When oral formulations of psychostimulants are used at recommended doses and frequencies, they are unlikely to yield effects consistent with abuse potential in patients with ADHD.

Retrieved 19 October Trends in Plant Science. Beyond 1 R ,2 S -ephedrine and 1 S ,2 S -pseudoephedrine, myriad other substituted amphetamines have important pharmaceutical applications.

Postgraduate Medical Journal. Archives of Toxicology. Annals of the New York Academy of Sciences.

JAMA Psychiatry. The Journal of Clinical Psychiatry. Acta Psychiatrica Scandinavica. Basal ganglia regions like the right globus pallidus, the right putamen, and the nucleus caudatus are structurally affected in children with ADHD.

These changes and alterations in limbic regions like ACC and amygdala are more pronounced in non-treated populations and seem to diminish over time from child to adulthood.

Treatment seems to have positive effects on brain structure. Ongoing research has provided answers to many of the parents' concerns, and has confirmed the effectiveness and safety of the long-term use of medication.

Among significantly improved outcomes, the largest effect sizes were found for combination treatment.

The greatest improvements were associated with academic, self-esteem, or social function outcomes. Pharmacology Biochemistry and Behavior.

Psychology Research and Behavior Management. Only one paper 53 examining outcomes beyond 36 months met the review criteria. Retrieved 12 November European Journal of Clinical Nutrition.

Cochrane Database of Systematic Reviews. Biological Psychiatry. The procognitive actions of psychostimulants are only associated with low doses.

Surprisingly, despite nearly 80 years of clinical use, the neurobiology of the procognitive actions of psychostimulants has only recently been systematically investigated.

Collectively, this evidence indicates that at low, clinically relevant doses, psychostimulants are devoid of the behavioral and neurochemical actions that define this class of drugs and instead act largely as cognitive enhancers improving PFC-dependent function.

In particular, in both animals and humans, lower doses maximally improve performance in tests of working memory and response inhibition, whereas maximal suppression of overt behavior and facilitation of attentional processes occurs at higher doses.

Journal of Cognitive Neuroscience. Specifically, in a set of experiments limited to high-quality designs, we found significant enhancement of several cognitive abilities.

The results of this meta-analysis Amphetamine has been shown to improve consolidation of information 0. Journal of Nuclear Medicine.

Dopamine acts in the nucleus accumbens to attach motivational significance to stimuli associated with reward. Pharmacological Reviews.

JS Online. Archived from the original on 15 August Retrieved 2 December NCAA Publications. National Collegiate Athletic Association.

Retrieved 8 October British Journal of Pharmacology. Clinics in Sports Medicine. In , Chandler and Blair 47 showed significant increases in knee extension strength, acceleration, anaerobic capacity, time to exhaustion during exercise, pre-exercise and maximum heart rates, and time to exhaustion during maximal oxygen consumption VO2 max testing after administration of 15 mg of dextroamphetamine versus placebo.

Most of the information to answer this question has been obtained in the past decade through studies of fatigue rather than an attempt to systematically investigate the effect of ADHD drugs on exercise.

Sports Medicine. In high-ambient temperatures, dopaminergic manipulations clearly improve performance. The distribution of the power output reveals that after dopamine reuptake inhibition, subjects are able to maintain a higher power output compared with placebo.

Dopaminergic drugs appear to override a safety switch and allow athletes to use a reserve capacity that is 'off-limits' in a normal placebo situation.

Frontiers in Integrative Neuroscience. Manipulations of dopaminergic signaling profoundly influence interval timing, leading to the hypothesis that dopamine influences internal pacemaker, or "clock," activity.

For instance, amphetamine, which increases concentrations of dopamine at the synaptic cleft advances the start of responding during interval timing, whereas antagonists of D2 type dopamine receptors typically slow timing; Depletion of dopamine in healthy volunteers impairs timing, while amphetamine releases synaptic dopamine and speeds up timing.

Frontiers in Physiology. Aside from accounting for the reduced performance of mentally fatigued participants, this model rationalizes the reduced RPE and hence improved cycling time trial performance of athletes using a glucose mouthwash Chambers et al.

Dopamine stimulating drugs are known to enhance aspects of exercise performance Roelands et al. This indicates that subjects did not feel they were producing more power and consequently more heat.

The authors concluded that the "safety switch" or the mechanisms existing in the body to prevent harmful effects are overridden by the drug administration Roelands et al.

Taken together, these data indicate strong ergogenic effects of an increased DA concentration in the brain, without any change in the perception of effort.

Southern Medical Journal. Rather they are intended to limit claims by pharmaceutical companies. International Programme on Chemical Safety.

Retrieved 24 June Journal of Affective Disorders. Tris Pharma, Inc. Retrieved 29 April Table 2. Archived from the original on 25 August Retrieved 24 December New England Journal of Medicine.

Archived from the original on 14 December Merck Manual for Health Care Professionals. Retrieved 8 May This study demonstrates that humans, like nonhumans, prefer a place associated with amphetamine administration.

These findings support the idea that subjective responses to a drug contribute to its ability to establish place conditioning.

Dialogues in Clinical Neuroscience. Despite the importance of numerous psychosocial factors, at its core, drug addiction involves a biological process: the ability of repeated exposure to a drug of abuse to induce changes in a vulnerable brain that drive the compulsive seeking and taking of drugs, and loss of control over drug use, that define a state of addiction.

Moreover, there is increasing evidence that, despite a range of genetic risks for addiction across the population, exposure to sufficiently high doses of a drug for long periods of time can transform someone who has relatively lower genetic loading into an addict.

Mount Sinai School of Medicine. Department of Neuroscience. Retrieved 9 February Substance-use disorder: A diagnostic term in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders DSM-5 referring to recurrent use of alcohol or other drugs that causes clinically and functionally significant impairment, such as health problems, disability, and failure to meet major responsibilities at work, school, or home.

Depending on the level of severity, this disorder is classified as mild, moderate, or severe. Addiction: A term used to indicate the most severe, chronic stage of substance-use disorder, in which there is a substantial loss of self-control, as indicated by compulsive drug taking despite the desire to stop taking the drug.

In the DSM-5, the term addiction is synonymous with the classification of severe substance-use disorder.

This is known to occur on many genes including fosB and c-fos in response to psychostimulant exposure. Chronic exposure to psychostimulants increases glutamatergic [signaling] from the prefrontal cortex to the NAc.

The Journal of General Physiology. Coincident and convergent input often induces plasticity on a postsynaptic neuron. The NAc integrates processed information about the environment from basolateral amygdala, hippocampus, and prefrontal cortex PFC , as well as projections from midbrain dopamine neurons.

Previous studies have demonstrated how dopamine modulates this integrative process. For example, high frequency stimulation potentiates hippocampal inputs to the NAc while simultaneously depressing PFC synapses Goto and Grace, KEGG Pathway.

Retrieved 31 October Most addictive drugs increase extracellular concentrations of dopamine DA in nucleus accumbens NAc and medial prefrontal cortex mPFC , projection areas of mesocorticolimbic DA neurons and key components of the "brain reward circuit".

Amphetamine achieves this elevation in extracellular levels of DA by promoting efflux from synaptic terminals.

Chronic exposure to amphetamine induces a unique transcription factor delta FosB, which plays an essential role in long-term adaptive changes in the brain.

Nature Reviews Neuroscience. The net result is gene activation and increased CDK5 expression. The net result is c-fos gene repression.

Clinical Psychopharmacology and Neuroscience. Magnesium Research. Similar to environmental enrichment, studies have found that exercise reduces self-administration and relapse to drugs of abuse Cosgrove et al.

There is also some evidence that these preclinical findings translate to human populations, as exercise reduces withdrawal symptoms and relapse in abstinent smokers Daniel et al.

In humans, the role of dopamine signaling in incentive-sensitization processes has recently been highlighted by the observation of a dopamine dysregulation syndrome in some patients taking dopaminergic drugs.

This syndrome is characterized by a medication-induced increase in or compulsive engagement in non-drug rewards such as gambling, shopping, or sex Evans et al.

Exercise has been proposed as a treatment for drug addiction that may reduce drug craving and risk of relapse.

Although few clinical studies have investigated the efficacy of exercise for preventing relapse, the few studies that have been conducted generally report a reduction in drug craving and better treatment outcomes Taken together, these data suggest that the potential benefits of exercise during relapse, particularly for relapse to psychostimulants, may be mediated via chromatin remodeling and possibly lead to greater treatment outcomes.

Frontiers in Neuroendocrinology. The postulate that exercise serves as an ideal intervention for drug addiction has been widely recognized and used in human and animal rehabilitation.

The limited research conducted suggests that exercise may be an effective adjunctive treatment for SUDs. In contrast to the scarce intervention trials to date, a relative abundance of literature on the theoretical and practical reasons supporting the investigation of this topic has been published.

Annual Review of Neuroscience. Progress in Neurobiology. Proceedings of the National Academy of Sciences.

Bibcode : PNAS.. Annals of Agricultural and Environmental Medicine. Nature Neuroscience. Nature Reviews. Journal of Psychoactive Drugs.

It has been found that deltaFosB gene in the NAc is critical for reinforcing effects of sexual reward. Pitchers and colleagues reported that sexual experience was shown to cause DeltaFosB accumulation in several limbic brain regions including the NAc, medial pre-frontal cortex, VTA, caudate, and putamen, but not the medial preoptic nucleus.

The Journal of Neuroscience. Pharmacologic treatment for psychostimulant addiction is generally unsatisfactory.

As previously discussed, cessation of cocaine use and the use of other psychostimulants in dependent individuals does not produce a physical withdrawal syndrome but may produce dysphoria, anhedonia, and an intense desire to reinitiate drug use.

Addiction Abingdon, England. Expert Review of Clinical Pharmacology. Despite concerted efforts to identify a pharmacotherapy for managing stimulant use disorders, no widely effective medications have been approved.

European Journal of Pharmacology. Existing data provided robust preclinical evidence supporting the development of TAAR1 agonists as potential treatment for psychostimulant abuse and addiction.

PLOS Medicine. Frontiers in Psychiatry. Physical Exercise There is accelerating evidence that physical exercise is a useful treatment for preventing and reducing drug addiction In some individuals, exercise has its own rewarding effects, and a behavioral economic interaction may occur, such that physical and social rewards of exercise can substitute for the rewarding effects of drug abuse.

The value of this form of treatment for drug addiction in laboratory animals and humans is that exercise, if it can substitute for the rewarding effects of drugs, could be self-maintained over an extended period of time.

Work to date in [laboratory animals and humans] regarding exercise as a treatment for drug addiction supports this hypothesis. Animal and human research on physical exercise as a treatment for stimulant addiction indicates that this is one of the most promising treatments on the horizon.

Merck Manual Home Edition. February Archived from the original on 17 February Retrieved 28 February Shoptaw SJ ed.

The prevalence of this withdrawal syndrome is extremely common Cantwell ; Gossop with The severity of withdrawal symptoms is greater in amphetamine dependent individuals who are older and who have more extensive amphetamine use disorders McGregor The first phase of this syndrome is the initial "crash" that resolves within about a week Gossop ;McGregor Amphetamine, dextroamphetamine, and methylphenidate act as substrates for the cellular monoamine transporter, especially the dopamine transporter DAT and less so the norepinephrine NET and serotonin transporter.

The mechanism of toxicity is primarily related to excessive extracellular dopamine, norepinephrine, and serotonin.

The Lancet. Retrieved 3 March Amphetamine use disorders Journal of Attention Disorders. The hyperthermia and the hypertension produced by high doses amphetamines are a primary cause of transient breakdowns in the blood-brain barrier BBB resulting in concomitant regional neurodegeneration and neuroinflammation in laboratory animals.

In animal models that evaluate the neurotoxicity of AMPH and METH, it is quite clear that hyperthermia is one of the essential components necessary for the production of histological signs of dopamine terminal damage and neurodegeneration in cortex, striatum, thalamus and hippocampus.

Direct toxic damage to vessels seems unlikely because of the dilution that occurs before the drug reaches the cerebral circulation.

Unlike cocaine and amphetamine, methamphetamine is directly toxic to midbrain dopamine neurons. Neurotoxicity Research.

Acta Medica Okayama. Molecular Pharmacology. Although the monoamine transport cycle has been resolved in considerable detail, kinetic knowledge on the molecular actions of synthetic allosteric modulators is still scarce.

This potentiates both the forward-transport mode i. Functional selectivity by conformational selection.

Expert Review of Neurotherapeutics. Zinc binds at In this context, controlled double-blind studies in children are of interest, which showed positive effects of zinc [supplementation] on symptoms of ADHD [,].

It should be stated that at this time [supplementation] with zinc is not integrated in any ADHD treatment algorithm.

Basal Ganglia. Despite the challenges in determining synaptic vesicle pH, the proton gradient across the vesicle membrane is of fundamental importance for its function.

Exposure of isolated catecholamine vesicles to protonophores collapses the pH gradient and rapidly redistributes transmitter from inside to outside the vesicle.

Amphetamine and its derivatives like methamphetamine are weak base compounds that are the only widely used class of drugs known to elicit transmitter release by a non-exocytic mechanism.

As substrates for both DAT and VMAT, amphetamines can be taken up to the cytosol and then sequestered in vesicles, where they act to collapse the vesicular pH gradient.

Frontiers in Systems Neuroscience. Three important new aspects of TAs action have recently emerged: a inhibition of firing due to increased release of dopamine; b reduction of D2 and GABAB receptor-mediated inhibitory responses excitatory effects due to disinhibition ; and c a direct TA1 receptor-mediated activation of GIRK channels which produce cell membrane hyperpolarization.

University of Paris. Retrieved 29 May AMPH also increases intracellular calcium Gnegy et al. Trends in Pharmacological Sciences.

AMPH and METH also stimulate DA efflux, which is thought to be a crucial element in their addictive properties [80], although the mechanisms do not appear to be identical for each drug [81].

See Laboratory Report. Room temperature. Reference ranges are provided as general guidance only. To interpret test results use the reference range in the laboratory report.

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